Search results for "MESH : Antioxidants"

showing 5 items of 5 documents

Do carotenoid-based sexual traits signal the availability of non-pigmentary antioxidants?

2006

SUMMARY Carotenoid-based signals are thought to be indicators of male quality because they must be obtained from the diet and might thus indicate the ability of individuals to gather high-quality food. However, carotenoids are also known to have important physiological functions as immunoenhancers and antioxidants, and, as such, carotenoid-based sexual traits have also been suggested to reflect the health and antioxidant status of their bearers. This last idea is based on the hypothesis that carotenoids that are allocated to sexual signals are no longer available for the detoxification system. Recently, this hypothesis has been challenged on the grounds that the antioxidant activity is not …

0106 biological sciencesMaleAntioxidantPhysiologymedicine.medical_treatmentMESH: Random AllocationMESH : LuteinMESH: BeakXanthophylls01 natural sciencesAntioxidantsRandom Allocationpolycyclic compounds[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisMESH: AnimalsFood scienceMESH : FinchesCarotenoidMESH: MelatoninMelatoninchemistry.chemical_classification0303 health sciencesSex CharacteristicsbiologyMESH : MelatoninPigmentationMESH : PigmentationBeakfood and beveragesPasserinecarotenoïdsBiochemistryMESH : AntioxidantsMESH : XanthophyllsMESH: Finchesmedicine.drugMESH: Sex CharacteristicsoxidationMESH : Malefree radicalsmacromolecular substances[ SDV.BBM.BM ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyAquatic ScienceMESH: Lutein010603 evolutionary biologyMESH: PigmentationMESH : Random AllocationMelatonin03 medical and health sciencessexual advertisementZeaxanthinsbiology.animalmedicineAnimalsMolecular BiologyZebra finchEcology Evolution Behavior and Systematics030304 developmental biologyMESH : Carotenoidsorganic chemicalsMESH: Antioxidantszebra finchLuteinMESH : Sex Characteristics[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyMESH: XanthophyllsCarotenoidsMESH: Malebiological factorsMESH : BeakchemistryInsect ScienceMESH: CarotenoidsAnimal Science and ZoologyMESH : AnimalsFinches[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/SymbiosisThe Journal of experimental biology
researchProduct

General oxidative stress during doxorubicin-induced cardiotoxicity in rats: Absence of cardioprotection and low antioxidant efficiency of alpha-lipoi…

2012

International audience; To evaluate the effects of alpha-lipoic acid (AL) in a model of doxorubicin (DOX)-induced cardiotoxicity, male Wistar rats were treated with DOX (1 mg/kg/d; 10 d) in combination or not with AL (50 mg/kg/d; 15 d). Plasma oxidative stress was determined by hydroperoxides (ROOH) and the ascorbyl radical/ascorbate ratio. One and two months later, the functional parameters of the hearts were determined in vivo by catheterization and cardiac oxidative stress was assessed by malonedialdehyde (MDA) and O₂*⁻ (dihydroethidium fluorescence) content in tissue. After two months, body weight was higher in the DOX-AL group than in DOX (+16%), but this was due to ascites. Histologic…

MaleMESH : Oxidative StressAntioxidantmedicine.medical_treatmentMESH : HematocritMESH : Thioctic AcidBiochemistryAntioxidants0302 clinical medicineSuperoxidesAscitic FluidMESH: AnimalsMESH : Body WeightComputingMilieux_MISCELLANEOUS0303 health sciencesThioctic AcidCumulative doseMESH: Heart DiseasesHeartGeneral Medicine3. Good healthMESH: Ascitic Fluid[SDV] Life Sciences [q-bio]030220 oncology & carcinogenesisMESH : Ascitic FluidMESH: Hydrogen PeroxideMESH : AntioxidantsMESH: Thioctic Acidmedicine.medical_specialtyCardiotonic AgentsCardiotoxinsMESH: Hematocrit03 medical and health sciencesMESH: Doxorubicin[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemIn vivoRats Wistar[ SDV ] Life Sciences [q-bio]MyocardiumMESH: AntioxidantsHydrogen PeroxideMESH: Cardiotonic AgentsMESH : Organ SizeMESH: Body WeightMESH: Heartcarbohydrates (lipids)EndocrinologyMESH: LiverMESH : SuperoxidesMESH: Organ Size[SDV]Life Sciences [q-bio]MESH : Cardiotonic AgentsAscorbic AcidMESH: Superoxidesmedicine.disease_causeMESH: EatingEatingpolycyclic compoundsMESH : MyocardiumMESH: Thiobarbituric Acid Reactive SubstancesMESH: Ascorbic AcidAntibiotics AntineoplasticMESH: Oxidative StressChemistryMESH : RatsOrgan SizeMESH : Antibiotics Antineoplastic[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemBiochemistryHematocritLiverMESH : Cardiotoxinsmedicine.drugMESH : EatingMESH: MyocardiumHeart DiseasesMESH: RatsMESH : MaleMESH : Thiobarbituric Acid Reactive SubstancesMESH : Rats WistarThiobarbituric Acid Reactive SubstancesContractilityMESH : HeartInternal medicinemedicineTBARSAnimalsMESH : DoxorubicinDoxorubicinMESH: Antibiotics AntineoplasticMESH : Ascorbic Acid030304 developmental biologyCardiotoxicityBody WeightMESH : LiverMESH : Heart DiseasesMESH: Rats WistarMESH: MaleRatsOxidative StressMESH: CardiotoxinsDoxorubicinMESH : AnimalsMESH : Hydrogen PeroxideOxidative stress
researchProduct

Is there a role for antioxidant carotenoids in limiting self-harming immune response in invertebrates?

2007

Innate immunity relies on effectors, which produce cytotoxic molecules that have not only the advantage of killing pathogens but also the disadvantage of harming host tissues and organs. Although the role of dietary antioxidants in invertebrate immunity is still unknown, it has been shown in vertebrates that carotenoids scavenge cytotoxic radicals generated during the immune response. Carotenoids may consequently decrease the self-harming cost of immunity. A positive relationship between the levels of innate immune defence and circulating carotenoid might therefore be expected. Consistent with this hypothesis, we show that the maintenance and use of the prophenoloxidase system strongly cor…

MaleantioxidantMESH : Immunity Natural[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunologyAntioxidantsMESH: Linear ModelsMESH: AmphipodaHemolymphMESH : Linear ModelsHemolymphMESH: AnimalsMESH : FemaleCarotenoidchemistry.chemical_classificationbiologyEffectorMonophenol Monooxygenasefood and beveragesProphenoloxidaseMESH : AmphipodaAgricultural and Biological Sciences (miscellaneous)MESH : Monophenol Monooxygenase[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyMESH : AntioxidantsFemaleGeneral Agricultural and Biological SciencesResearch ArticleMESH: Monophenol MonooxygenaseMESH : Maleimmune costsecological immunologyMESH : Hemolymph[ SDV.BBM.BM ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyImmune systemImmunityAnimalsAmphipodaMESH: Immunity NaturalMESH : CarotenoidsInnate immune systemMESH: HemolymphMESH: Antioxidants[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologybiochemical phenomena metabolism and nutritionbiology.organism_classificationCarotenoidsImmunity InnateMESH: MaleGammarus pulexchemistryImmunologyMESH: CarotenoidsLinear ModelsbacteriaMESH : AnimalsMESH: Female
researchProduct

Grape seed and skin extract reduces pancreas lipotoxicity, oxidative stress and inflammation in high fat diet fed rats.

2016

IF 2.326; International audience; Obesity is related to an elevated risk of diabetes and the mechanisms whereby fat adversely affects the pancreas are poorly understood. We studied the effect of a high fat diet (HFD) on pancreas steatosis, oxidative stress and inflammation as well as the putative protection afforded by grape seed and skin extract (GSSE). HFD induced body weight gain, without affecting insulinemia, nor glycemia and dropped adiponectemia. HFD also provoked the ectopic deposition of cholesterol and triglyceride, and an oxidative stress characterized by increased lipoperoxidation and carbonylation, inhibition of antioxidant enzyme activities such as CAT, GPx and SOD, depletion …

MaleMESH : Oxidative Stress0301 basic medicineMESH: InflammationAntioxidantmedicine.medical_treatmentMESH: Grape Seed Extractmedicine.disease_causeAntioxidantschemistry.chemical_compound0302 clinical medicineMESH: ObesityVitisMESH: AnimalsMESH : Pancreas2. Zero hungerMESH: Oxidative StressMESH : Grape Seed ExtractMESH : RatsMESH : Diet High-Fatfood and beveragesGeneral MedicineMESH: Pancreas3. Good healthLipotoxicity030220 oncology & carcinogenesisSeedsMESH : AntioxidantsMESH : Obesitymedicine.symptommedicine.medical_specialtyGrape seed and skin extractMESH: RatsMESH : MaleInflammationBiologyDiet High-FatMESH : Rats WistarMESH: VitisMESH : Vitis03 medical and health sciencesDiabetes mellitusInternal medicinemedicineAnimalsObesityRats WistarPancreasPharmacologyInflammationMESH : InflammationGrape Seed ExtractTriglycerideCholesterolMESH: Antioxidants[ SDV.SP.PHARMA ] Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologyMESH: Rats Wistarmedicine.diseaseMESH : SeedsMESH: MaleRatsMESH: Diet High-Fat030104 developmental biologyEndocrinologychemistryMESH: SeedsOxidative stress[SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologyMESH : AnimalsSteatosisOxidative stress
researchProduct

Effect of oxidative stress on UDP-glucuronosyltransferases in rat astrocytes.

2012

WOS:000309170300003; International audience; The present work reports data regarding effects of an induced oxidative stress on the mainly expressed isoforms of UDP-glucuronosyltransferases (UGTs) in the brain. UGT1A6 and UGT1A7 expression and enzymatic activities toward the 1-naphthol were analyzed in rat cultured astrocytes following the exposure for 48 h to redox-cycling xenobiotic compounds such as quinones and bipyridinium ions. The expression of NADPH:cytochrome P450 reductase and NAD(P)H:quinone oxidoreductase 1 (NQO1) was also investigated. Oxidative stress induced significant deleterious changes in astrocyte morphology, decreased cell viability and inhibited catalytic function of UG…

MESH : Oxidative StressMESH : RNA MessengerAntioxidantTranscription Geneticmedicine.medical_treatmentToxicologyNAD(P)H:quinone oxidoreductase 1MESH: GlucuronosyltransferaseAntioxidantsSubstrate SpecificityRats Sprague-Dawley0302 clinical medicineMESH: NADPH-Ferrihemoprotein ReductaseMESH: GlucuronidesNAD(P)H Dehydrogenase (Quinone)MESH : CatalysisMESH: AnimalsMESH : NAD(P)H Dehydrogenase (Quinone)GlucuronosyltransferaseCells Culturedchemistry.chemical_classificationMESH : Cell Survival0303 health sciencesMESH : Substrate SpecificityMESH : Animals NewbornCytochrome P450 reductaseGeneral MedicineMESH: Cell SurvivalMESH: Pyridinium CompoundsMESH : AntioxidantsMESH: Cells CulturedOxidative phosphorylationGene Expression Regulation EnzymologicMESH : QuinonesMESH : Glucuronides03 medical and health sciencesRNA MessengerCell ShapeNADPH-Ferrihemoprotein ReductaseMESH : Oxidation-ReductionMESH : Pyridinium CompoundsMESH: NaphtholsMESH : GlucuronosyltransferaseMESH: AntioxidantsMESH: CatalysischemistryOxidative stressAstrocytesReactive Oxygen Species030217 neurology & neurosurgeryMESH: Oxidation-ReductionTime Factors[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Reactive Oxygen SpeciesNADPH:cytochrome P450 reductasePyridinium CompoundsNaphtholsMESH: Rats Sprague-DawleyProtein oxidationmedicine.disease_causeMESH: Animals NewbornMESH: NAD(P)H Dehydrogenase (Quinone)Protein CarbonylationMESH : OxidantsMESH: OxidantsMelatoninMESH: MelatoninMESH: Oxidative StressMESH : MelatoninMESH : RatsMESH: Gene Expression Regulation EnzymologicQuinonesMESH: Reactive Oxygen SpeciesOxidantsBiochemistryMESH : Protein CarbonylationOxidation-ReductionUDP-glucuronosyltransferaseMESH : Time FactorsMESH: Protein CarbonylationMESH: RatsCell SurvivalMESH : NaphtholsBiologyCatalysisMESH: QuinonesMESH : Gene Expression Regulation EnzymologicGlucuronidesMESH : Cells CulturedmedicineAnimalsMESH: Cell Shape030304 developmental biologyMESH: RNA MessengerReactive oxygen speciesMESH: Transcription GeneticMESH: Time FactorsMESH : AstrocytesMESH : Transcription GeneticNAD(P)H Dehydrogenase (Quinone)MESH : Rats Sprague-DawleyRatsMESH: AstrocytesAnimals NewbornMESH : NADPH-Ferrihemoprotein ReductaseMESH: Substrate SpecificityMESH : AnimalsNAD+ kinaseMESH : Cell Shape[SDV.AEN]Life Sciences [q-bio]/Food and NutritionOxidative stress
researchProduct